headshot of Partha Dutta

Partha Dutta

Associate Professor
Dutta Lab Google Scholar Linkedin Twitter Bioengineering Department


Florentin, J., Zhao, J., Tai, Y.Y., Sun, W., Ohayon, L.L., O'Neil, S.P., Arunkumar, A., Zhang, X., Zhu, J., Al Aaraj, Y., Watson, A., Sembrat, J., Rojas, M., Chan, S.Y., & Dutta, P. (2022). Loss of Amphiregulin drives inflammation and endothelial apoptosis in pulmonary hypertension. LIFE SCIENCE ALLIANCE, 5(11), e202101264.Life Science Alliance, LLC. doi: 10.26508/lsa.202101264.

Johny, E., & Dutta, P. (2022). Left Coronary Artery Ligation: A Surgical Murine Model of Myocardial Infarction. JOVE-JOURNAL OF VISUALIZED EXPERIMENTS, (186).MyJove Corporation. doi: 10.3791/64387.

Ohayon, L., Zhang, X., & Dutta, P. (2021). The role of extracellular vesicles in regulating local and systemic inflammation in cardiovascular disease. PHARMACOLOGICAL RESEARCH, 170, 105692.Elsevier BV. doi: 10.1016/j.phrs.2021.105692.

Vasamsetti, S.B., Coppin, E., Zhang, X., Florentin, J., Koul, S., Gotberg, M., Clugston, A.S., Thoma, F., Sembrat, J., Bullock, G.C., Kostka, D., St Croix, C.M., Chattopadhyay, A., Rojas, M., Mulukutla, S.R., & Dutta, P. (2020). Apoptosis of hematopoietic progenitor-derived adipose tissue-resident macrophages contributes to insulin resistance after myocardial infarction. SCIENCE TRANSLATIONAL MEDICINE, 12(553).American Association for the Advancement of Science (AAAS). doi: 10.1126/scitranslmed.aaw0638.

Coppin, E., Florentin, J., Vasamsetti, S.B., Arunkumar, A., Sembrat, J., Rojas, M., & Dutta, P. (2018). Splenic hematopoietic stem cells display a pre-activated phenotype. IMMUNOLOGY AND CELL BIOLOGY, 96(7), 772-784.Wiley. doi: 10.1111/imcb.12035.

Florentin, J., Coppin, E., Vasamsetti, S.B., Zhao, J., Tai, Y.Y., Tang, Y., Zhang, Y., Watson, A., Sembrat, J., Rojas, M., Vargas, S.O., Chan, S.Y., & Dutta, P. (2018). Inflammatory Macrophage Expansion in Pulmonary Hypertension Depends upon Mobilization of Blood-Borne Monocytes. JOURNAL OF IMMUNOLOGY, 200(10), 3612-3625.The American Association of Immunologists. doi: 10.4049/jimmunol.1701287.

Vasamsetti, S.B., Florentin, J., Coppin, E., Stiekema, L.C.A., Zheng, K.H., Nisar, M.U., Sembrat, J., Levinthal, D.J., Rojas, M., Stroes, E.S.G., Kim, K., & Dutta, P. (2018). Sympathetic Neuronal Activation Triggers Myeloid Progenitor Proliferation and Differentiation. IMMUNITY, 49(1), 93-+.Elsevier BV. doi: 10.1016/j.immuni.2018.05.004.

Bracamonte-Baran, W., Florentin, J., Zhou, Y., Jankowska-Gan, E., Haynes, W.J., Zhong, W., Brennan, T.V., Dutta, P., Claas, F.H.J., van Rood, J.J., & Burlingham, W.J. (2017). Modification of host dendritic cells by microchimerism-derived extracellular vesicles generates split tolerance. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 114(5), 1099-1104.Proceedings of the National Academy of Sciences. doi: 10.1073/pnas.1618364114.

Dutta, P., Gomez, D., & Gladwin, M.T. (2017). Do BRD(4) S of a Feather Flock Together? How an Inflammation-Driven Epigenetic Regulator May Link Pulmonary Hypertension and Coronary Artery Disease. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 37(8), 1428-1430.Ovid Technologies (Wolters Kluwer Health). doi: 10.1161/ATVBAHA.117.309632.

Florentin, J., & Dutta, P. (2017). Origin and production of inflammatory perivascular macrophages in pulmonary hypertension. CYTOKINE, 100, 11-15.Elsevier BV. doi: 10.1016/j.cyto.2017.08.015.

Florentin, J., Zhao, J., Tai, Y.Y., Vasamsetti, S.B., O'Neil, S.P., Kumar, R., Arunkumar, A., Watson, A., Sembrat, J., Bullock, G.C., Sanders, L., Kassa, B., Rojas, M., Graham, B.B., Chan, S.Y., & Dutta, P. (2021). Interleukin-6 mediates neutrophil mobilization from bone marrow in pulmonary hypertension. In CELLULAR & MOLECULAR IMMUNOLOGY, 18(2), (pp. 374-384).Springer Science and Business Media LLC.China. doi: 10.1038/s41423-020-00608-1.

Sager, H., Dutta, P., Dahlman, J., Hulsmans, M., Courties, G., Sun, Y., Heidt, T., Borodovsky, A., Fitzgerald, K., Libby, P., Langer, R., Weissleder, R., Swirski, F., Anderson, D., & Nahrendorf, M. (2016). Nanoparticle-enabled RNAi in endothelial cells attenuates inflammation in atherosclerosis and acute myocardial infarction. In EUROPEAN HEART JOURNAL, 37, (p. 989).